Max in WT synaptoneurosomes, suggesting that Src signaling may very well be downregulated in KI synapses. Alternatively, our capacity to rescue SERT functionality in KI midbrain synaptoneurosomes from the inhibition of FAK suggests elevated FAK signaling downstream with the Pro32Pro33 mutant, as verified by elevated pFAK localization in 5-HT synapses. https://pro33login34433.bloggazzo.com/31470264/pro33-secrets